Book Contents

Histopathology and Cytology of
Poultry Diseases
By Ivan Dinev, DVM, PhD


CHOLANGIOHEPATITIS

Fig 1. Liver. Multiple outgrown bile
ducts, forming a granuloma structure
with a central necrosis. H/E, Bar = 35
µm.

Fig 1. Liver. Multiple outgrown bile ducts, forming a granuloma structure with a central necrosis. H/E, Bar = 35 µm.

 
Fig. 2. Liver. A biliary stasis and a huge
amount of Gram-positive organisms in
bile ducts. Gram staining, Bar = 25 µm.

Fig. 2. Liver. A biliary stasis and a huge amount of Gram-positive organisms in bile ducts. Gram staining, Bar = 25 µm.

 
Fig. 3. Liver. Pericanalicular biliary necroses.
H/E, Bar = 50 µm.

Fig. 3. Liver. Pericanalicular biliary necroses. H/E, Bar = 50 µm.

 
Fig. 4. Liver. Fibrosed and thickened
wall of a bile duct, filled with Grampositive
microorganisms after the
occurred inflammatory and necrotic
changes. H/E, Bar = 35 µm.

Fig. 4. Liver. Fibrosed and thickened wall of a bile duct, filled with Grampositive microorganisms after the occurred inflammatory and necrotic changes. H/E, Bar = 35 µm.

 
Fig. 5. Hepatosis in the liver of a broiler
chicken. Proliferation of bile ducts
and fibrous tissue, that have infiltrated
the portal zones through mononuclear
cells. H/E, Bar = 25 µm.

Fig. 5. Hepatosis in the liver of a broiler chicken. Proliferation of bile ducts and fibrous tissue, that have infiltrated the portal zones through mononuclear cells. H/E, Bar = 25 µm.

 
This book is protected by the copyright law. The reproduction, imitation or distribution of the book in whole or in part, in any format (electronic, photocopies etc.) without the prior consent, in writing, of copyright holders is strictly prohibited.
© 2000 - 2024 - Global Ag Media. All Rights Reserved | No part of this site may be reproduced without permission.