Book Contents

Histopathology and Cytology of
Poultry Diseases
By Ivan Dinev, DVM, PhD


PULMONARY HYPERTENSION (ASCITES) SYNDROME IN BROILER CHICKENS

Fig. 1. Separation of intermyocardial
fibres by a moderate to extensive
oedema of loose connective tissue.
Subepicardial and myocardial haemorrhages
of a various size. H/E, Bar
= 50 µm.

Fig. 1. Separation of intermyocardial fibres by a moderate to extensive oedema of loose connective tissue. Subepicardial and myocardial haemorrhages of a various size. H/E, Bar = 50 µm.

 
Fig. 2. Lung. Hyperaemia, haemorrhages
and oedema. A possible finding
is the appearance of of bone and
cartilage foci (co) among the pulmonary
parenchyma. H/E, Bar = 30 µm.

Fig. 2. Lung. Hyperaemia, haemorrhages and oedema. A possible finding is the appearance of of bone and cartilage foci (co) among the pulmonary parenchyma. H/E, Bar = 30 µm.

 
Fig. 3. Liver sinusoids are enlarged
and overfilled with blood. Supplementary
hepatic findings are the
oedema and atrophic degenerative
lesions. H/E, Bar = 50 µm.

Fig. 3. Liver sinusoids are enlarged and overfilled with blood. Supplementary hepatic findings are the oedema and atrophic degenerative lesions. H/E, Bar = 50 µm.

 
Fig. 4. Compression atrophy of liver
parenchyma secondary to congested
sinusoids resulting from the passive
venous hyperaemia. H/E, Bar = 30
µm.

Fig. 4. Compression atrophy of liver parenchyma secondary to congested sinusoids resulting from the passive venous hyperaemia. H/E, Bar = 30 µm.

 
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