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    Book Contents

    1. Histopathology and Cytology Associated with Bacterial Diseases
    2. Histopathology and Cytology Associated with Viral Diseases
    3. Histopathology and Cytology Associated with Neoplastic Diseases
    4. Histopathology and Cytology Associated with Parasitic Diseases
    5. Histopathology and Cytology Associated with Mycotic Diseases
    6. Histopathology and Cytology Associated with Deficiency Diseases
    7. Histopathology and Cytology Associated with Other Diseases
    Histopathology and Cytology of
    Poultry Diseases
    By Ivan Dinev, DVM, PhD


    VITAMIN E DEFICIENCY

    Fig. 1. Histologically, colliquation necroses are observed, mainly in the white brain substance of corpus medularis, appearing as brighter foci, extensively vacuolized and with a reticular structure. H/E, Bar = 30 µm.

    Fig. 1. Histologically, colliquation necroses are observed, mainly in the white brain substance of corpus medularis, appearing as brighter foci, extensively vacuolized and with a reticular structure. H/E, Bar = 30 µm.

     
    Fig. 2. Haemorrhages of a various size are observed among the necrotic tissue. H/E, Bar = 35 µm.

    Fig. 2. Haemorrhages of a various size are observed among the necrotic tissue. H/E, Bar = 35 µm.

     
    Fig. 3. Haemorrhages and multiple thrombosed blood vessels in the cerebelum. H/E, Bar = 100 µm.

    Fig. 3. Haemorrhages and multiple thrombosed blood vessels in the cerebelum. H/E, Bar = 100 µm.

     
    Fig. 4. Muscular dystrophy, pectoral musculature, hen. Hyaline degeneration of muscle fibres (waxy degeneration). Swol len homogeneous muscle fibres, having lost their striated pattern, with increased eosinophilia. Centrally, necrobiotic lesions resulting in muscle cell degradation (Zenker’s necrosis). H/E, Bar = 25 µm.

    Fig. 4. Muscular dystrophy, pectoral musculature, hen. Hyaline degeneration of muscle fibres (waxy degeneration). Swol len homogeneous muscle fibres, having lost their striated pattern, with increased eosinophilia. Centrally, necrobiotic lesions resulting in muscle cell degradation (Zenker’s necrosis). H/E, Bar = 25 µm.

     
    Fig. 5. Late stage of muscular dystrophy, thigh muscle, chicken. Marked acidophilia of muscle cells and beginning of regenerative reparative processes, organization. H/E, Bar = 35 µm.

    Fig. 5. Late stage of muscular dystrophy, thigh muscle, chicken. Marked acidophilia of muscle cells and beginning of regenerative reparative processes, organization. H/E, Bar = 35 µm.

     
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