Book Contents

Histopathology and Cytology of
Poultry Diseases
By Ivan Dinev, DVM, PhD


IONOPHORE TOXICITY CHICKENS

Fig. 1. & Fig. 2. Transverse/londitudinal
cross-section, thigh muscle, chicken,
after intoxication with high doses
maduramycin. Enhanced eosinophilia
and denenerative necrobiotic lesions
of muscle fibres. H/E, Bar = 40 µm.

Fig. 1. & Fig. 2. Transverse/londitudinal cross-section, thigh muscle, chicken, after intoxication with high doses maduramycin. Enhanced eosinophilia and denenerative necrobiotic lesions of muscle fibres. H/E, Bar = 40 µm.

 
Fig. 1. & Fig. 2. Transverse/londitudinal
cross-section, thigh muscle, chicken,
after intoxication with high doses
maduramycin. Enhanced eosinophilia
and denenerative necrobiotic lesions
of muscle fibres. H/E, Bar = 40 µm.

 
Fig. 3. Longitudinal cross-section,
thigh muscle, chicken, after intoxication
with high doses maduramycin.
Appearance of macrophages and initial
organization of necrotic detritus
occuring after muscle fibre breakdown.
H/E, Bar = 50 µm.

Fig. 3. Longitudinal cross-section, thigh muscle, chicken, after intoxication with high doses maduramycin. Appearance of macrophages and initial organization of necrotic detritus occuring after muscle fibre breakdown. H/E, Bar = 50 µm.

 
Fig. 4. Inflammatory, degenerative
necrobiotic lesions and urate deposits
in renal tubules. H/E, Bar = 25 µm.

Fig. 4. Inflammatory, degenerative necrobiotic lesions and urate deposits in renal tubules. H/E, Bar = 25 µm.

 
Fig. 5. Urate cylinders (arrow) and retention
cysts (rc) in kidneys. H/E, Bar
= 50 µm.

Fig. 5. Urate cylinders (arrow) and retention cysts (rc) in kidneys. H/E, Bar = 50 µm.

 
Fig. 6. Degenerative necrobiotic lesions
of the liver. H/E, Bar = 50 µm.

Fig. 6. Degenerative necrobiotic lesions of the liver. H/E, Bar = 50 µm.

 
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